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development of IAA was thought to be probably as a result of oxidase deamination of NMT (Barker et al., 1980), but was later on proven being also partly by direct deamination by MAO (Barker et al., 1982). Pretreatment Along with the MAO-I iproniazid in rat entire brain homogenates inhibited IAA formation by 83%, NMT and DMT-NO formation were inhibi